Steroid induced diabetes in pregnancy

Diagnosis Index entries containing back-references to :

  • Diabetes, diabetic (mellitus) (sugar) ICD-10-CM Diagnosis Code Type 2 diabetes mellitus without complications
      2016 2017 2018 Billable/Specific Code Questionable As Admission Dx
    • with
      • retinopathy ICD-10-CM Diagnosis Code Type 2 diabetes mellitus with unspecified diabetic retinopathy without macular edema
          2016 2017 2018 Billable/Specific Code
        • with macular edema
    • type 2 ICD-10-CM Diagnosis Code Type 2 diabetes mellitus without complications
        2016 2017 2018 Billable/Specific Code Questionable As Admission Dx
      • with
        • retinopathy ICD-10-CM Diagnosis Code Type 2 diabetes mellitus with unspecified diabetic retinopathy without macular edema
            2016 2017 2018 Billable/Specific Code
          • with macular edema

Riddle me this? How do two doctors send a diabetic home with steriods for an undisclosed condtion? And never did they mention and changes I might need to be aware of, being a diabetic. Not to menation, the fact that they couldn’t figure out or even consider psorisis now that I have learned more about it, it’s pretty common. I’m not a doctor and I wasn’t aware of this disease. What I have become aware of, is if you catch it early you can take steps to minimize the breakout hence pain. I’m considering taking further action.

Steroid-induced osteoporosis (SIOP) is osteoporosis arising due to use of glucocorticoids (steroid hormones) - analogous to Cushing's syndrome and involving mainly the axial skeleton. The synthetic glucocorticoid prescription drug prednisone is a main candidate after prolonged intake. Bisphosphonates are beneficial in reducing the risk of vertebral fractures. [1] Some professional guidelines recommend prophylactic calcium and vitamin D supplementation in patients who take the equivalent of more than 30 mg hydrocortisone ( mg of prednisolone), especially when this is in excess of three months. [2] [3] The use of thiazide diuretics, and gonadal hormone replacement has also been recommended, with the use of calcitonin, bisphosphonates, sodium fluoride or anabolic steroids also suggested in refractory cases. [4] Alternate day use may not prevent this complication. [5]

Cells of the zona fasciculata and zona reticularis lack aldosterone synthase (CYP11B2) that converts corticosterone to aldosterone, and thus these tissues produce only the weak mineralocorticoid corticosterone. However, both these zones do contain the CYP17A1 missing in zona glomerulosa and thus produce the major glucocorticoid, cortisol. Zona fasciculata and zona reticularis cells also contain CYP17A1, whose 17,20-lyase activity is responsible for producing the androgens, dehydroepiandrosterone (DHEA) and androstenedione. Thus, fasciculata and reticularis cells can make corticosteroids and the adrenal androgens, but not aldosterone.

Steroid induced diabetes in pregnancy

steroid induced diabetes in pregnancy

Cells of the zona fasciculata and zona reticularis lack aldosterone synthase (CYP11B2) that converts corticosterone to aldosterone, and thus these tissues produce only the weak mineralocorticoid corticosterone. However, both these zones do contain the CYP17A1 missing in zona glomerulosa and thus produce the major glucocorticoid, cortisol. Zona fasciculata and zona reticularis cells also contain CYP17A1, whose 17,20-lyase activity is responsible for producing the androgens, dehydroepiandrosterone (DHEA) and androstenedione. Thus, fasciculata and reticularis cells can make corticosteroids and the adrenal androgens, but not aldosterone.

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