For the record, I'll add my story: I was prescribed 1000mg of Cipro per day for seven days, for a UTI a year ago. I was a healthy 24-year-old (nothing about me was a risk factor for the tendinopathy adverse reaction). After the first 2000mg, all of my tendons over my body felt like they were on fire. Felt like they could suddenly snap at any moment. I searched the web and found out that tendon damage was a side effect of Cipro. I was livid as I wasn't warned about this from the doctor or the pharmacist. Went back to the clinic to change my antibiotics to Amoxicillin, and the idiot doctor insisted I keep taking the Cipro as stopping the course was "dangerous" -- this, when I told her I was getting tendonitis from the Cipro.
Over one year later, I'm still suffering daily from tendon and joint pain all over my body, with new pains appearing all the time. I'm convinced the damage is permanent, and am thinking about suing.
The point is (and all the others who have responded to you already have emphasized this), it is very clear that you need the help of those who deal with difficult uveitis cases day in and day out (link to list of uveitis specialists). Unfortunately, it appears that you must be prepared to travel to get the help of such a person. But do it you must; and the consultant whom you choose will undoubtedly want to see you again, too. But he or she will also want to work with an ophthalmologist and perhaps a second specialist (., a rheumatologist or a hematologist) in an effort to give to you the care that you need to preserve the vision you have, and hopefully to get back at least a little of what you have lost.
When activated macrophages start to secrete IL-1, which synergistically with CRH increases ACTH,  T-cells also secrete glucosteroid response modifying factor (GRMF), as well as IL-1; both increase the amount of cortisol required to inhibit almost all the immune cells.  Immune cells then assume their own regulation, but at a higher cortisol setpoint. The increase in cortisol in diarrheic calves is minimal over healthy calves, however, and falls over time.  The cells do not lose all their fight-or-flight override because of interleukin-1's synergism with CRH. Cortisol even has a negative feedback effect on interleukin-1  —especially useful to treat diseases that force the hypothalamus to secrete too much CRH, such as those caused by endotoxic bacteria. The suppressor immune cells are not affected by GRMF,  so the immune cells' effective setpoint may be even higher than the setpoint for physiological processes. GRMF affects primarily the liver (rather than the kidneys) for some physiological processes.